Desmethylprodine-02 Technology / Science in Pax Imperia - WASC21 | World Anvil
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Desmethylprodine-02

Desmethylprodine-02 is a modified version of an experimental drug, under development by the Oktavgradian military. While the original version of the drug, tested on Humans, was created as an analgesic, it was discovered that an impurity in its synthesis, tested on Dea, causes inhibited ability of theriantropy. It is considered a silent technological breakthrough.  

Disovery

When first synthesized, Desmethylprodine was meant to be developed and sold as a safe, pain-relieving drug. After a short period of testing on volunteers, it was noted that while some percent of its users responded positively to the drug, a large amount of specifically Dea subjects reported feeling incapacitated with intense sickness. It was discovered that a common impurity produced during the synthesis of the drug causes intense short-term effects on Dea, as opposed to any other species of users. Further studies and development of Desmethylprodine have been officially ceased as the military was instructed to step in in light of this discovery.

Access & Availability

Information about the development of this new drug has not been made public and is being kept a secret by the Oktavgradian military for the time being. As a safety precaution and as to bypass a wave of concern regarding the ethics of its use, only the research personnel and high-ranking military officials know of its existence.

Utility

The produced impurity of the drug acts as a competitive inhibitor of the enzymes responsible for the changes in a Dea's body. While under the influence of the drug, in addition to the inhibited ability of theriantropy, the subjects may experience reduced pain, drowsiness, low blood pressure and vomiting.
The drug is injected and its effects may last between 3-7 hours.

The long term effects of the drug injections have proven to negatively affect the dopaminergic neurons in the brain, affecting the nervous and motor system. These effects have not been studied any further.

The enzyme inhibition can be reversed if overcome with a sufficiently high substrate concentration, in order to out-compete it.
Legal status
Not in circulation (Schedule I)
Elimination half-life
3-7 hours (dose-dependent)

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